Liver failure Prevention and Prognosis

Prevention for Acute Liver failure
Acute liver failure have a higher mortality rate, so should be as much as possible to prevention its occurrence. Clinical medicine can do is to observe the adverse effects on the liver. For example: tuberculosis with rifampicin, amine or sulfur Isonicotinic pyrazinamide treatment, etc., should check the blood transaminase, bilirubin, etc., such as changes in liver function was found to be in time to change the drug. Implementation of the larger surgical trauma of the surgery, patients should pay attention to the preoperative liver function, particularly the original liver cirrhosis, hepatitis, jaundice, hypoproteinemia, such as disease, and should be fully prepared. Should avoid use of narcotic drug hepatotoxicity. Postoperative course of surgery and as far as possible to prevent hypoxia, hypotension or shock, infection, so as to avoid damage to liver cells; after the condition of patients to continue to monitor the liver function and maintain good respiratory cycle, anti-infection and the maintenance of nutritional metabolism, liver a good role.

Prognosis for Acute Liver failure
The survival rate of acute liver failure is different that due to circumstances and causes. In young patients from acetaminophen poisoning or hepatitis caused by the survival rate of up to 50% of those who, over the age of 40 patients and caused by certain drugs hepatitis, the survival rate of less than 10% mortality rate after liver transplantation in situ reduced to 20% ~ 30%, 1-year survival rate of 55% to 80%. As a result of liver transplantation to be effective in saving the lives of patients, the prognosis for patients with poor liver transplantation should be timely and therefore indicators of poor prognosis that is, the indications for liver transplantation. Radiation on the film show that the poor prognosis of cerebral edema, liver transplantation should be apparent radiation-chip prior to cerebral edema. Hepatitis associated with fulminant hepatic failure patients on admission if the apparent performance of intracranial hypertension, prothrombin time> 100s and age> 50 years old, the incidence of hepatic encephalopathy with jaundice appears intervals> 7 days have prompted a poor prognosis.