G6PD

Treating the symptoms associated with G6PD deficiency is usually as simple as removing the trigger, treating the illness or infection or stopping the use of a certain drug, blood transfusions, dialysis, splenectomy. G6PD deficiency is prevented by avoiding fava beans, oxidant drugs, and oxidant chemicals.

The diagnosis of Glucose-6-phosphate dehydrogenase deficiency is generally suspected when patients from certain ethnic groups develop anemia, jaundice and symptoms of hemolysis, especially when there is a positive family history.

The most significant consequence of G6PD deficiency is hemolytic anemia, symptoms are jaundice, dark urine, abdominal pain, back pain, lowered red blood cell count, and elevated bilirubin.

Glucose-6-phosphatase dehydrogenase (G6PD) deficiency is a genetic condition. It is caused by one copy of a defective G6PD gene, also be triggered by certain drugs (aspirin, antimalarial, antibiotics, dapsone), infections, fava beans.

G6PD deficiency is an inherited disease in which the body have no enough of the enzyme glucose-6-phosphate dehydrogenase which causes premature destruction of red blood cells.