Atherosclerosis is the most common arteriosclerosis diseases, which is characterized by arterial disease begin with intima. General first accumulation of lipids and complex carbohydrates, hemorrhage and thrombosis, fibrous tissue hyperplasia and calcinosis, as well as the gradual transformation and calcification of the middle artery. Lesions often involving large and medium-sized elastic and muscular arteries, such as, the disease mainly involving the large-scale and medium-sized muscular artery elasticity to the aortic, coronary and cerebral artery found. Once the development of blocked arteries enough cavity, the arterial supply to the tissues or organs or ischemic necrosis. Atherosclerosis were more common in men over the age of 40 and women after menopause. The disease is often accompanied by hypertension, hypercholesterolemia, or diabetes. See more mental.
Atherosclerosis is involved from the aorta to the body of the circulatory system diseases intima. Characterized by the formation of arterial intimal plaque. Each of the different components of plaque. Lipid atherosclerotic plaque is the basic ingredient. Intimal thickening is not, strictly speaking atherosclerotic plaque, but mechanical damage to the intima of an adaptive response.
Atherosclerosis is the main cause of death. Origin of atherosclerosis in childhood and continued to progress, usually in middle-aged or elderly symptoms. As a result of atherosclerotic plaque lipid showed necrotic tissue and the first poly, so often that atherosclerosis is a degenerative disease. Now believe that this disease is the result of factors, first of all lesions in the smooth muscle cells, macrophages and T lymphocytes together; followed, including collagen, elastic fibers and connective tissue matrix proteins and polysaccharides, such as smooth muscle cells hyperplasia; and third, lipid, mainly containing cholesterol crystal and free cholesterol and connective tissue. Atherosclerotic plaque lipid and connective tissue content of the decision and whether the stability of plaques lead to acute ischemic events.