Causes of Kawasaki disease is infection, pathogens such as EB virus, retrovirus, Streptococcus, Propionibacterium, and environmental pollution or chemicals, autoimmune disorder.

Kawasaki disease, muco-cuta-meous lymph node syndrome (MCLS), is acute febrile rash disease in children characterized by systemic vasculitis, include four phases.

Surgical Treatment for Hepatic injury is hemostasis, removal of fragmentation in liver tissue and placement of peritoneal abteilung, debridement, hepatic artery ligation, hepatectomy.

Diagnosis for Hepatic injury (liver injury) is peritoneal puncture, Determination of red blood cells, hemoglobin and hematocrit, B-ultrasonic, X-ray examination, radionuclide liver scan.

The most common complications of Hepatic injury (liver injury) is infection, and biliary fistula, secondary hemorrhage and acute liver, kidney failure.

Symptoms of Hepatic injury divided into true liver laceration that signs is excessive bleeding, shock; subcapsular liver laceration is right upper quadrant pain, central liver laceration is upper gastrointestinal bleeding.

Treatment for Hepatic hemangioma is surgery, interventional therapy, sclerotherapy, resection of hemangioma, orthotopic liver transplantation, radiation therapy.

Causes of Hepatic injury (liver injury) is traumatic injury, include open and closed injury, such as knife stab injury, gunshot wounds, blindgut wound and penetrating wound.

Hepatic injury (Liver injury) are more common and serious injury in abdominal trauma, occur intra-abdominal bleeding or bile leakage, hemorrhagic shock or bile peritonitis.

Hepatic hemangioma Symptoms is liver pain, hepatomegaly, mass and pressure, loss of appetite, dyspepsia; Complication is gastrointestinal bleeding, hemobilia.

Diagnosis for Hepatic hemangioma (liver hemangioma) include B-Ultra, CT scan, Hepatic arteriography, Laparoscopic examination, X-ray tests.

Causes of Hepatic hemangioma (liver hemangioma) is dysthymia, long-term smoking, alcohol, fatty food, irritant spicy food, and other risk factors.

Hepatic hemangioma (liver hemangioma) is the majority of cavernous hemangioma, is a common benign tumor of the liver, can occur at any age, but often occur in women.

The treatment of acute cholangitis use surgical treatment to remove bile duct obstruction factors, to ensure smooth flow of biliary drainage.

Acute cholangitis symptoms is the upper abdominal discomfort and pain, chills, little fever and jaundice, and sometimes attack or was colic.

The diagnosis of acute cholangitis is depond on B-ultrasound that can be found in the expansion of common bile duct wall thickening, and sometimes also show bile duct stones or roundworm.

Acute cholangitis is syndrome on an obstruction of the biliary tree most commonly from a gallstone, characterized by fever, jaundice, and abdominal pain that develops as a result of stasis and infection in the biliary tract.

NRDS is prevented by prenatal prevention that use adreno cortical hormone, post-natal prevention that take infant with pulmonary surfactant, and combinated prevention.

Treatment for NRDS is infant nursing, ensure fluids and nutrition supply, close patent ductus arteriosus, antibiotics, oxygen therapy, mechanical ventilation, PS Alternative medicine.

Diagnosis for NRDS include X-ray Imaging, laboratory tests, prenatal amniotic fluid and post partum endotracheal substance test, biochemical examination, foam test.

Symptoms of NRDS is respiratory difficulty, groaning, irregular breathing, occasionally apnea, pale or gray due to hypoxia, cyanosis, in 6-12 hours after birth.

Causes of NRDS is the lack of pulmonary surfactant (PS), risk factors is Premature Infant, Infant of diabetic pregnant women, Intrauterine distress and birth asphyxia.

Neonatal respiratory distress syndrome (NRDS), is the newborn infant after a brief of natural breathing, and subsequent progressive dyspnea, cyanosis, acute respiratory distress and respiratory failure.

Prognosis of ARDS is related with rescue measures, primary disease and complications; prevention of ARDS include closely observed, intensive care for high-risk patients.

Treatment of ARDS is correct hypoxia, oxygen therapy, mechanical ventilation, PEEP, elimination of pulmonary edema, medication such as adrenocorticotropic hormone.

Diagnosis for ARDS is chest X-ray examination, arterial blood gas analysis, Pulmonary function test, Pulmonary vascular permeability and hemodynamic determination.

Symptoms of ARDS include respiratory distress, chest contracting, inspiratory dyspnea, cyanosis, anxiety, extensive stromal invasion of lungs, expansion of azygos vein, pleural effusion.

Pathogenesis of ARDS is the migration, aggregation and release of inflammatory cells, alveolar capillary damage and increased permeability, ARDS have an effects on pulmonary respiration.

Causes of ARDS is serious shock, severe trauma, fat embolism fractures, serious infections, inhalation of irritant gas, oxygen poisoning, result in pulmonary interstitial, alveolar edema, and hypoxemia.

ARDS is acute respiratory failure syndrome characterized by diffuse pulmonary capillary injury, pulmonary edema, hyaline membrane, atelectasis, progressive hypoxemia.