Treatment of HUS is supportive, include management of fluids and electrolytes, blood transfusion, plasma exchange (plasma remove and replace), kidney dialysis.
HUS is diagnosed by CBC, blood clotting tests, urinalysis, stool cultures; Abnormalities include anemia, thrombocytopenia, uremia, blood and protein in urine and E. coli O157:O7.
Complications of HUS are renal failure, hypertension, hematuria, TTP, neurologic dysfunction, gastrointestinal, pancreatitis, cardiac dysfunction and more.
Symptoms of HUS are abdominal cramping, vomiting, profuse bloody, watery diarrhea, weakness, fatigue, low or no urine output, unexplained bruises or bleeding, paleness, yellow skin.
HUS often is caused by an infection with E. coli O157:H7 or other bacteria, and viral infection such pneumonia bacteria, autoimmune disease such AIDS, blood infection, medications such mitomycin C, pregnancy and postpartum.
E coli O157:H7 is a strain of the bacterium Escherichia coli, It causes foodborne illness, colitis and bloody diarrhea by producing a toxin called Shiga toxin, which damages the intestines.
Hemolytic uremic syndrome, HUS, is a condition characterized by the destruction of red blood cells and kidney failure that is associated with microangiopathic hemolytic anemia and thrombocytopenia.
Hemoglobin variants is Hemoglobin S, Hemoglobin C, Hemoglobin E, Hemoglobin H, Hemoglobin Barts, Compound Heterozygous Conditions include Hemoglobin C, Hemoglobin S-C, Hemoglobin D and Hemoglobin E disease.
Hemoglobinopathy is a group of inherited disorders involving abnormal structure of the hemoglobin molecule. These disorders include hemoglobin C disease , hemoglobin S-C disease, sickle cell anemia, and various types of thalassemia.
Hemangioblastoma is treated by surgical resection, remove tumor and stereotactic radiosurgery, reduce tumor size or to retard growth.
Hemangioblastoma is diagnosed by neurological examination, computed tomography (CT) or magnetic resonance imaging (MRI) scan and cerebral angiography, complete neural axis imaging.
Causes of hemangioblastomas is inherited VHL gene as an autosomal dominant trait, Symptoms of hemangioblastoma are depend on the location of the tumor, common include headache, loss of coordination, imbalance, nausea and vomiting.
Hemangioblastoma is a benign, highly vascular tumor that most frequently occur in the cerebellar hemispheres and vermis, brain stem, spinal cord, it arise sporadically or associated with von Hippel Lindau disease.
Treating the symptoms associated with G6PD deficiency is usually as simple as removing the trigger, treating the illness or infection or stopping the use of a certain drug, blood transfusions, dialysis, splenectomy. G6PD deficiency is prevented by avoiding fava beans, oxidant drugs, and oxidant chemicals.
The diagnosis of Glucose-6-phosphate dehydrogenase deficiency is generally suspected when patients from certain ethnic groups develop anemia, jaundice and symptoms of hemolysis, especially when there is a positive family history.
The most significant consequence of G6PD deficiency is hemolytic anemia, symptoms are jaundice, dark urine, abdominal pain, back pain, lowered red blood cell count, and elevated bilirubin.
Glucose-6-phosphatase dehydrogenase (G6PD) deficiency is a genetic condition. It is caused by one copy of a defective G6PD gene, also be triggered by certain drugs (aspirin, antimalarial, antibiotics, dapsone), infections, fava beans.
G6PD deficiency is an inherited disease in which the body have no enough of the enzyme glucose-6-phosphate dehydrogenase which causes premature destruction of red blood cells.
Treatment for eosinophilia are based on their identify causes, allergic and connective tissue disorders may be amenable to corticosteroid treatment. Parasitic and fungal infections can be worsened or disseminated by use of steroids and should be ruled out.
Eosinophilia due to asthma appear symptoms such as wheezing and breathlessness, parasitic infections lead to abdominal pain, diarrhoea, fever, cough and rashes. Medicine reactions lead to skin rashes. Other symptoms of eosinophilia can include weight loss, night sweats, lymph node enlargement, numbness and nerve damage. Eosinophilia is diagnosed by blood test, skin biopsy, CT scans.
Eosinophilia causes include a wide range of conditions. Its commonest causes are allergic diseases such as asthma and hay fever, parasitic infections such as intestinal worms. It can also occur in relation to common skin diseases, medicine reactions.
Eosinophilia is an abnormal increase in the eosinophils (type of white blood cell) in blood or body tissues. Eosinophils are produced in the bone marrow existing normally in the bloodstream and gut lining, which helps the body fight infection from parasitic organisms.
Sideroblastic anemia is a rare blood disorders characterized by the bone marrow's inability to manufacture normal red blood cells. Sideroblastic anemias is one of the principal types of iron-utilization anemias that are usually part of a myelodysplastic syndrome, producing a normocytic-normochromic anemia with high RBC distribution width or a microcytic-hypochromic anemia, particularly with increased serum iron, ferritin, and transferrin saturation. Three categories of sideroblastic anemia are: hereditary, acquired or idiopathic.
Folic acid deficiency anemia may be suspected from general findings from a complete medical history and physical examination. In addition, several tests can be performed to confirm the diagnosis.
Alcoholism is a common cause of folic acid deficiency anemia. Alcohol interferes with the absorption of several nutrients especially the B vitamins, folate.
Folic acid deficiency anemia, an abnormally low level of folic acid (one of the B vitamins), results in anemia characterized by red blood cells that are large in size but few in number, which can cause a type of anemia known as megaloblastic (pernicious) anemia.
Specific treatment for hemolytic anemia will be determined by your physician based on by several factors including your age, overall health, medical history, causes of hemolytic anemia.
Some people with autoimmune hemolytic anemia may have no symptoms, especially when the destruction of red blood cells is mild and develops gradually. When the destruction is more severe or rapid, most common symptoms of hemolytic anemia may include...
Hemolytic anemia is a blood disorder in which the red blood cells are destroyed prematurely. The cells are broken down at a faster rate than the bone marrow can produce new cells. The term for destruction of red blood cells is hemolysis.
Megaloblastic anemia, also called pernicious anemia, is a blood disorder in which there is anemia with red blood cells that are larger than normal.